Information on Cadasil

CADASIL (cerebral autosomal dominantmight also have these health problems.
arteriopathy with subcortical infarcts andGenetic counseling is the process of providing
leukoencephalopathy) is the most common formindividuals and families with information on the
of hereditary stroke disorder, and is thought tonature, inheritance, and implications of genetic
be caused by mutations of the Notch 3 gene ondisorders to help them make informed medical
chromosome 19. The most common clinicaland personal decisions. The following section deals
manifestations are migraine headaches andwith genetic risk assessment and the use of
transient ischemic attacks or strokes, whichfamily history and genetic testing to clarify genetic
usually occur between 40 and 50 years of age,status for family members. This section is not
although MRI is able to detect signs of the diseasemeant to address all personal, cultural, or ethical
years prior to clinical manifestation of disease.issues that individuals may face or to substitute
Cerebral autosomal arteriopathy with subcorticalfor consultation with a genetics professional.
infarcts and leukoencephalopathy (CADASIL) isThe disease primarily affects small blood vessels
characterized by recurrent subcortical ischemicin the white matter of the brain. A mutation in the
strokes and dementia caused by mutations in theNotch3 gene alters the muscular walls in these
Notch3 gene. In Drosophila melanogaster, Notchsmall arteries. CADASIL is characterized by
signaling has a pleiotropic effect, affecting mostmigraine headaches and multiple strokes
tissues of the organism during development.progressing to dementia. Other symptoms include
Biochemical, histochemical, molecular, and geneticwhite matter lesions throughout the brain,
analyses were performed on muscle biopsycognitive deterioration, seizures, vision problems,
specimens and fibroblasts obtained from patientsand psychiatric problems such as severe
of a Spanish family with CADASIL. Additionaldepression and changes in behavior and
biochemical and molecular analyses of thepersonality. Individuals may also be at higher risk
N(55e11) mutant of D. melanogaster wereof heart attack. Symptoms and disease onset
performed.vary widely, with signs typically appearing in the
An abnormality in the muscle cells surroundingmid-30s. Some individuals may not show signs of
these blood vessels (vascular smooth musclethe disease until later in life.
cells) gradually destroys these cells. The resultingThere are several different methods of diagnosing
blood vessel damage can cause migraines andCADASIL. One of the most common methods is
other impairments of normal brain function. Laterto take a skin biopsy, and examine it with a
in life, the damaged blood vessels can causetechnique called electron microscopy, to look for
reduced blood flow to various tissues in the bodythe characteristic accumulations of granular
(ischemia). Although the severity of symptomsmaterial (called granular osmiophilic material, or
varies among those affected, people withGOM) commonly seen in CADASIL. This can
CADASIL typically have more than one stroke inpositively diagnose CADASIL, though a negative
their lifetime. Recurrent strokes can progressivelyresult on this test does not necessarily mean that
damage the brain, causing loss of intellectualthe disease is not present. Additionally, a skin
function (dementia). CADASIL is not associatedbiopsy can be tested for the accumulation of
with the common risk factors for stroke andNotch3, using a molecule that specifically detects
heart attack, such as high blood pressure and highthis protein.
cholesterol, although some affected individuals